ZOOHCC - 501: Molecular Biology (Theory)



Unit 4: Translation 











    Inhibitors of protein synthesis



    Inhibitors of protein synthesis are molecules that interfere with the
    process of translation, which is the process by which genetic information in
    mRNA is used to synthesize a protein. Translation involves the coordinated
    action of many different proteins and RNA molecules, and inhibitors of
    protein synthesis can target various steps in this process.



    There are several types of inhibitors of protein synthesis, including
    antibiotics and chemical inhibitors. Antibiotics are natural or synthetic
    compounds that are produced by microorganisms and are used to treat
    bacterial infections. Many antibiotics target bacterial protein synthesis by
    inhibiting specific steps in translation, such as the binding of
    aminoacyl-tRNA to the ribosome or the formation of peptide bonds between
    amino acids.



    Chemical inhibitors are synthetic compounds that can be designed to
    specifically target different components of the translation machinery. They
    can be useful for studying the mechanisms of translation and for developing
    new drugs to treat bacterial infections. However, they can also have toxic
    effects on eukaryotic cells and can lead to unwanted side effects if used
    improperly.



    In general, inhibitors of protein synthesis can be very effective in
    treating bacterial infections, but it is important to use them judiciously
    to avoid the development of antibiotic resistance and other negative
    consequences.



    Explanation



    Inhibitors of protein synthesis are molecules that interfere with the
    process of translation, the synthesis of proteins from mRNA. These
    inhibitors can be broadly classified into two categories:



    Antibiotics: Antibiotics are natural or synthetic compounds that are
    produced by microorganisms and are used to treat bacterial infections. Many
    antibiotics target bacterial protein synthesis by inhibiting specific steps
    in translation. Examples of antibiotics that inhibit protein synthesis
    include:



    Aminoglycosides: These antibiotics bind to the small subunit of the
    ribosome and interfere with the accuracy of translation.



    Tetracyclines: These antibiotics bind to the small subunit of the ribosome
    and prevent the attachment of aminoacyl-tRNA to the ribosome.



    Macrolides: These antibiotics bind to the large subunit of the ribosome and
    inhibit the elongation phase of translation.



    Chemical inhibitors: Chemical inhibitors are synthetic compounds that can
    be designed to specifically target different components of the translation
    machinery. Examples of chemical inhibitors of protein synthesis
    include:



    Cycloheximide: This chemical inhibitor binds to the large subunit of the
    ribosome and blocks the translocation step of translation.



    Puromycin: This chemical inhibitor mimics an aminoacyl-tRNA and is
    incorporated into the growing polypeptide chain, causing premature
    termination of translation.



    Chloramphenicol: This chemical inhibitor binds to the large subunit of the
    ribosome and inhibits the peptidyl transferase activity, preventing the
    formation of peptide bonds.



    In general, inhibitors of protein synthesis can be very useful in studying
    the mechanisms of translation and in developing new drugs to treat bacterial
    infections. However, they can also have toxic effects on eukaryotic cells
    and can lead to unwanted side effects if used improperly.



    Difference between prokaryotic and eukaryotic translation



    Prokaryotic and eukaryotic cells differ in many ways, including the process
    of translation, which is the synthesis of proteins from mRNA. Here are 10
    differences between prokaryotic and eukaryotic translation:





    1. Ribosome size: Prokaryotic ribosomes are smaller (70S) than eukaryotic
      ribosomes (80S).



    2. Initiation factors: Prokaryotes have fewer initiation factors than
      eukaryotes.



    3. Shine-Dalgarno sequence: Prokaryotic mRNA contains a Shine-Dalgarno
      sequence that helps to initiate translation, whereas eukaryotic mRNA does
      not.



    4. mRNA splicing: Eukaryotic mRNA may undergo splicing before translation,
      removing introns and joining exons. Prokaryotic mRNA does not undergo
      splicing.



    5. Polycistronic mRNA: Prokaryotic mRNA can be polycistronic, meaning it
      codes for multiple proteins. Eukaryotic mRNA is typically monocistronic,
      coding for a single protein.



    6. Post-translational modifications: Eukaryotic proteins may undergo
      post-translational modifications, such as glycosylation or
      phosphorylation, that are not found in prokaryotes.



    7. Translation location: In prokaryotes, transcription and translation can
      occur simultaneously, whereas in eukaryotes, transcription occurs in the
      nucleus and translation occurs in the cytoplasm.



    8. Elongation factors: Prokaryotes and eukaryotes use different elongation
      factors in the process of translation.



    9. Antibiotics: Some antibiotics target prokaryotic ribosomes and can be
      used to treat bacterial infections. Eukaryotic ribosomes are not affected
      by these antibiotics.



    10. Translation regulation: Eukaryotic translation is more tightly regulated
      than prokaryotic translation, with multiple mechanisms for controlling the
      rate of protein synthesis.